Abstract
We Examined a 70-Year-Old Woman Who Became Aphasic after a Left Thalamic Infarction. Computed Tomographic Scan Showed Injury that Was Largely Limited to the Ventral Anterior and Rostral Ventral Lateral Thalamic Nuclei. Speech Was Characterized by Reduced Voice Volume, Impaired Auditory and Reading Comprehension, Perseverations, Intermittent Use of Jargon, Fluctuations in the Ability to Perform Confrontation Naming, Extraneous Intrusions, Verbal Paraphasia, Intact Repetition Skills, and Fluent Speech that Was Laconic But Grammatically Correct. We Propose that the Deficits after Left Thalamic Injury Can Be Grouped into the Following Four Large Clusters: Extrapyramidal Deficits (Decreased or Fading Voice Volume), Deficits in Lexical Access (Anomia, Verbal Paraphasia), Deficits in Vigilance (Neologisms, Intrusions, Fluctuating Performance, Jargon, Perseverations), and Comprehension Defects. © 1984, American Medical Association. All Rights Reserved.
Recommended Citation
P. B. Gorelick et al., "Aphasia after Left Thalamic Infarction," Archives of Neurology, vol. 41, no. 12, pp. 1296 - 1298, JAMA Neurology, Jan 1984.
The definitive version is available at https://doi.org/10.1001/archneur.1984.04050230082026
Department(s)
Chemistry
International Standard Serial Number (ISSN)
1538-3687; 0003-9942
Document Type
Article - Journal
Document Version
Citation
File Type
text
Language(s)
English
Rights
© 2023 JAMA Neurology, All rights reserved.
Publication Date
01 Jan 1984
PubMed ID
6497734
